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Aortic insufficiency and arterial hypertension can lead to the development of congestive heart failure.

In addition to high sustained arterial hypertension, clinical manifestations of heart damage may be due to the expansion of the aortic root with blood regurgitation on the aortic valve and narrowing of Robaxin arteries in the region of the orifices and subepicardial segments, which is manifested by angina pectoris, up to the development of myocardial infarction.

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Significantly rarer complications are aortic dissection and rupture. Less commonly, patients are concerned about high intermittent claudication and vague abdominal pain.

In the diagnosis of Takayasu's disease, Doppler study of blood flow in the branches of the aorta and echocardiography are of great importance. Confirmation of the diagnosis allows X-ray contrast aortography, in which areas of narrowing of Robaxin and places of origin of its branches of various lengths are determined due to thickening of their walls, up to complete occlusion. AnEurysmatic expansion of the thoracic aorta is rare and is not characteristic of this disease.

Laboratory signs of active inflammation, as well as its clinical manifestations, are usually absent in the stage of aortic stenosis.

robaxin Therapy

Takayasu's disease is characterized by a slowly progressing course over many years. The prognosis noticeably worsens with the appearance of robaxin complications, primarily from the brain, heart, and kidneys.

If there are signs of systemic inflammation, glucocorticosteroids are prescribed, sometimes in combination with cytostatic which are immunosuppressants. If at the same time there are already ischemic manifestations, such therapy can help to reduce them.

In the absence of general inflammatory signs, the use of Methocarbamol drugs is not indicated. Although such patients are often prescribed antiplatelet agents and anticoagulants of direct and indirect action to improve blood flow, they are generally not very effective.

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The only effective method of treating occlusive lesions of the aorta and its branches are surgical reconstructive operations, including percutaneous angioplasty.

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Giant cell arteritis is characterized by immune granulomatous inflammation predominantly of the medium-sized arteries of the head and neck, with the greatest constancy of the temporal arteries, with the development of their stenosis.

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In less than half of the patients, the aorta and its large branches, mainly extending from the arch, are also involved in the process.

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It is possible to develop an aneurysm of the ascending aorta, aortic insufficiency, and occasionally - dissection and rupture of the aorta.

According to its clinical picture in such cases, giant cell arteritis may have much in common with Takayasu's disease, differing from it in a significantly lower incidence of damage to the descending thoracic and abdominal aorta and arterial hypertension.

Mostly people over the age of 50 are ill, and men and women are equally common. As a rule, pronounced signs of general inflammation are noted: fever, weakness, sweating, weight loss, a significant increase in ESR and anemia.